
A common gut bacterium produces a molecule that slows cholesterol production in the liver. When researchers gave the molecule to mice prone to artery disease, the animals developed roughly half as much fatty plaque as untreated controls.
The molecule acts on the same enzyme targeted by statins, the widely used cholesterol-lowering drugs. That connection offers a concrete explanation for something harder to untangle: how bacteria living in the intestine might influence the health of arteries elsewhere in the body.
Researchers at institutions including Sun Yat-sen University and Shenzhen ...
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