Ceftriaxone partially restores glutamate dynamic deficits around Aβ deposits.
New research from the Djavad Mowafaghian Centre for Brain Health at UBC has found a way to partially restore brain cell communication around areas damaged by plaques associated with Alzheimer’s disease. The findings, published this week in Nature Communications, demonstrate a possible target and a potential drug treatment to reduce damage to the brain that occurs in the early stages of Alzheimer’s disease. Using Ceftriaxone, an FDA-approved antibiotic used to treat bacterial infections, researchers were able to reduce synaptic disruption and clear the lines of neuronal communication in mice.
Amyloid plaques of -amyloid deposits develop in brain regions of patients with Alzheimer’s disease, These plaques are link...
Read More
Recent Comments